IL-35 inhibits acute graft-versus-host disease in a mouse model

Xiao Hui Zhang*, Yi Zhou, Jia Min Zhang, Shi Yuan Zhou, Min Wang, Ru Feng, Fer Er Feng, Qian Ming Wang, Xiao Lu Zhu, Xiao Su Zhao, Meng Lv, Yuan Kong, Ying Jun Chang, Xiao Jun Huang

*Corresponding author for this work

Research output: Contribution to journalArticlepeer-review

12 Citations (Scopus)

Abstract

Acute graft-versus-host disease (aGVHD) is a serious complication of allogeneic hematopoietic stem cell transplantation (allo-HSCT). Our previous study found that the novel anti-inflammatory cytokine IL-35 could suppress aGVHD in patients after allo-HSCT. In this study, we used C57BL/6 (B6, H-2b) mice as donors and (B6 × DBA/2) F1 (BDF1, H-2b × d) mice as recipients to create a model of aGVHD and explore the relationship between IL-35 and aGVHD. The mice receiving IL-35 survived longer than did the control mice. We observed that treatment with IL-35 and RAPA could reduce the incidence of aGVHD. Additionally, this treatment inhibited intestinal and thymic epithelial cell apoptosis and liver infiltration by the donor T-cells, thereby ameliorating the enteropathy and liver injury caused by aGVHD. We found that IL-35 and RAPA also markedly suppressed TNF-α and IL-17A expression and enhanced IFN-γ expression in the intestine and liver. We measured Tregs in spleen and found that IL-35 and RAPA treatment expanded the number of Tregs in spleen. We found that the phosphorylation of STAT1 and STAT4 were inhibited in mice with aGVHD. In contrast, STAT1 and STAT4 were phosphorylated when the mice were treated with IL-35. IL-35 may have therapeutic potential in the treatment of aGVHD after allo-HSCT.

Original languageEnglish
Pages (from-to)383-392
Number of pages10
JournalInternational Immunopharmacology
Volume29
Issue number2
DOIs
Publication statusPublished - 1 Dec 2015
Externally publishedYes

Keywords

  • Acute graft-versus-host disease
  • Bone marrow transplantation
  • IL-35
  • Rapamycin

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